TPMT*47 is assigned no function based on limited evidence. TPMT*47 is characterized by a four base pair deletion in exon II (c.21_24delACTT) resulting in a frameshift of the coding sequence and the creation of a premature stop codon, which is predicted to undergo nonsense-mediated decay. The variant effect predictors, CADD and PhD-SNPg, predict that c.21_24del impairs protein function.